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Carlos Puebla ● Profesor Asistente
![](https://www.uoh.cl/instituto-ciencias-de-la-salud/wp-content/uploads/sites/26/2024/01/Carlos-Puebla-4.png)
Doctorado en Ciencias Biológicas, mención en Ciencias Fisiológicas – Pontificia Universidad Católica de Chile
Bioquímico – Pontificia Universidad Católica de Chile
Descripción
Carlos obtuvo el título de Bioquímico en la Pontificia Universidad Católica de Chile, PUC (Santiago). Además, tiene un Magister en Ciencias Biológicas y un Doctorado en Ciencias Biológicas, mención en Ciencias Fisiológicas, ambos grados obtenidos en la misma universidad. Su formación doctoral fue apoyada por CONICYT (Comisión Nacional de Investigación Científica y Tecnológica) con dos becas: Beca de Doctorado Nacional y Tesis de Apoyo Doctoral. Carlos tiene una formación postdoctoral en el Departamento de Fisiología de la Facultad de Ciencias Biológicas en la PUC, donde obtuvo una beca posdoctoral de FONDECYT (Fondo Nacional para el Desarrollo Científico y Tecnológico). Actualmente, el Dr. Puebla es Profesor Asistente asociado al Instituto de Ciencias de la Salud de la Universidad de O'Higgins (Rancagua).
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- REVISTA Frontiers in Public Health
- 2024
Community strategies for health promotion and prevention of chronic non-communicable diseases with a focus on physical activity and nutrition: the URO/FOCOS study protocol
• Matías Monsalves-Álvarez • María Teresa Solis-Soto • María Soledad Burrone • Alejandro A. Candia • Emilio Jofré-Saldía
- REVISTA Microorganisms
- 2023
Microbiota profile of the nasal cavity according to lifestyles in healthy adults in Santiago, Chile
• Daniela Toro-Ascuy • Juan P. Cardenas • Francisco Zorondo • Damariz González • Evelyn Silva-Moreno
- REVISTA Frontiers in Physiology
Editorial Free Fatty Acids as Signaling Molecules: Role of Free Fatty Acid Receptors and CD36
• Carlos Puebla • Eugenia Morselli • Naim Akhtar Khan • Mauricio A. Retamal •
- REVISTA Frontiers in Physiology
Use of short chain fatty acids for the recovery of the intestinal epithelial barrier affected by bacterial toxins
• Diliana Perez R • Eduardo Karahanian • Carlos Puebla • Katherine García •
- REVISTA Nature Communications
Active acetylcholine receptors prevent the atrophy of skeletal muscles and favor reinnervation
• Bruno A. Cisterna • Aníbal A. Vargas • Carlos Puebla • Paola Fernández • Rosalba Escamilla
- REVISTA Neural Regeneration Research
Pannexin 1-based channels activity as a novel regulator of multiple sclerosis progression
• Fernando C. Ortiz • Carlos Puebla •
- REVISTA Biochimica Biophysica Acta - Molecular Basis of Disease
De novo expression of functional connexins 43 and 45 hemichannels increases sarcolemmal permeability of skeletal myofibers during endotoxemia.
• Luis A. Cea • Elisa Balboa • Aníbal A. Vargas • Carlos Puebla • María C Brañes
- REVISTA Frontiers in Cellular Neuroscience
Role of Connexin-Based Gap Junction Channels in Communication of Myelin Sheath in Schwann Cells
• Bruno A. Cisterna • Pablo Arroyo • Carlos Puebla •
- REVISTA Frontiers in Physiology
Regulation of Connexin-Based Channels by Fatty Acids
• Carlos Puebla • Mauricio A. Retamal • Rodrigo Acuña • Juan C. Sáez •
- REVISTA Cellular and Molecular Life Sciences
Fast skeletal myofibers of mdx mouse, model of Duchenne muscular dystrophy, express connexin hemichannels that lead to apoptosis
• Luis A. Cea • Carlos Puebla • Bruno A. Cisterna • Rosalba Escamilla • Aníbal A. Vargas
- REVISTA Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease
Dexamethasone-induced muscular atrophy is mediated by functional expression of connexin-based hemichannels
• Luis A. Cea • Elisa Balboa • Carlos Puebla • Aníbal A. Vargas • Bruno A. Cisterna
- REVISTA Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease
Connexin hemichannels explain the ionic imbalance and lead to atrophy in denervated skeletal muscles
• Bruno A. Cisterna • Aníbal A. Vargas • Carlos Puebla • Juan C. Sáez •
- REVISTA Biochimica et Biophysica Acta (BBA) - Molecular and Cell Biology of Lipids
Linoleic acid permeabilizes gastric epithelial cells by increasing connexin 43 levels in the cell membrane via a GPR40- and Akt-dependent mechanism
• Carlos Puebla • Bruno A. Cisterna • Daniela P. Salas • Fernando Delgado-López • Paul D Lampe
- REVISTA Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease
Role of Akt and Ca2+ on cell permeabilization via connexin43 hemichannels induced by metabolic inhibition
• Daniela P. Salas • Carlos Puebla • Paul D Lampe • Sergio Lavandero • Juan C. Sáez
- REVISTA Experimental Biology and Medicine
Featured Article: Dexamethasone and rosiglitazone are sufficient and necessary for producing functional adipocytes from mesenchymal stem cells
• David Contador • Fernando Ezquer • Maximiliano Espinosa • Martha Arango-Rodriguez • Carlos Puebla
- REVISTA Frontiers in Cell and Developmental Biology
Pannexin channels mediate the acquisition of myogenic commitment in C2C12 reserve cells promoted by P2 receptor activation
• Manuel A. Riquelme • Luis A. Cea • José L. Vega • Carlos Puebla • Aníbal A. Vargas
- REVISTA Proceedings of the National Academy of Sciences
De novo expression of connexin hemichannels in denervated fast skeletal muscles leads to atrophy
• Luis A. Cea • Bruno A. Cisterna • Carlos Puebla • Marina Frank • Xavier F. Figueroa
- REVISTA The Journal of Membrane Biology
Connexin- and Pannexin-Based Channels in Normal Skeletal Muscles and Their Possible Role in Muscle Atrophy
• Luis A. Cea • Manuel A. Riquelme • Bruno A. Cisterna • Carlos Puebla • José L. Vega
- REVISTA PLOS ONE
Gestational Diabetes Reduces Adenosine Transport in Human Placental Microvascular Endothelium, an Effect Reversed by Insulin
• Carlos Salomón • Francisco Westermeier • Carlos Puebla • Pablo Arroyo • Enrique Guzmán-Gutierrez
- REVISTA Placenta
Review: Differential placental macrovascular and microvascular endothelial dysfunction in gestational diabetes
• Luis Sobrevia • Fernando Abarzua • Jyh K. Nien • Carlos Salomón • Francisco Westermeier
- REVISTA Current Vascular Pharmacology
Functional Link Between Adenosine and Insulin: A Hypothesis for Fetoplacental Vascular Endothelial Dysfunction in Gestational Diabetes
• Enrique Guzmán-Gutierrez • Fernando Abarzua • Cristian Belmar • Jyh K. Nien • Marco A. Ramirez
- REVISTA Diabetes
Insulin Restores Gestational Diabetes Mellitus-Reduced Adenosine Transport Involving Differential Expression of Insulin Receptor Isoforms in Human Umbilical Vein Endothelium
• Francisco Westermeier • Carlos Salomón • Marcelo González • Carlos Puebla • Enrique Guzmán-Gutierrez
- REVISTA Cardiovascular Research
TGF-?1 inhibits expression and activity of hENT1 in a nitric oxide-dependent manner in human umbilical vein endothelium
• José L. Vega • Carlos Puebla • Rodrigo Vásquez • Marcelo Farías • Julio Alarcón
- REVISTA Cardiovascular Research
Nitric oxide reduces SLC29A1 promoter activity and adenosine transport involving transcription factor complex hCHOP-C/EBP? in human umbilical vein endothelial cells from gestational diabetes
• Marcelo Farías • Carlos Puebla • Francisco Westermeier • Miguel J. Jo • Marçal Pastor-Anglada
- REVISTA Current Vascular Pharmacology
Potential Cell Signalling Mechanisms Involved in Differential Placental Angiogenesis in Mild and Severe Pre-Eclampsia
• Carlos Escudero • Carlos Puebla • Francisco Westermeier • Luis Sobrevia •
- REVISTA Current Vascular Pharmacology
Equilibrative Nucleoside Transporters in Fetal Endothelial Dysfunction in Diabetes Mellitus and Hyperglycaemia
• Francisco Westermeier • Carlos Puebla • José L. Vega • Marcelo Farías • Carlos Escudero
- REVISTA Journal of Cellular Physiology
HighD-glucose reducesSLC29A1 promoter activity and adenosine transport involving specific protein 1 in human umbilical vein endothelium
• Carlos Puebla • Marcelo Farías • Marcelo González • Andrea Vecchiola • Claudio Aguayo
- REVISTA Journal of Cellular Physiology
Nitric oxide reduces adenosine transporter ENT1 gene (SLC29A1) promoter activity in human fetal endothelium from gestational diabetes
• Marcelo Farías • Rody San Martín • Carlos Puebla • Jeremy D. Pearson • Javier F. Casado
- Enero 2023
- Abril 2022
- Diciembre 2021
- Abril 2019
- Marzo 2019
- Noviembre 2016
Investigador/a Responsable
- Marzo 2015
- Octubre 2012